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Learn MoreWe have employed gene expression profiling in order to identify targets of transcriptional response to stress in resting mouse Swiss 3T3 fibroblasts, either untreated (control) or treated with anisomycin for 3 or 6 hours to induce the p38/MAP kinase pathway.In order determine transcriptional effects dependent on MSK1/2 kinase activity, H89 inhibitor was used in the study. SOURCE: Anna Sawicka (anna.sawicka@univie.ac.at) - Seiser Medical University of Vienna/MFPL
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