PLX314716

GSE95822: Length-independent telomere damage drives cardiomyocyte senescence

Organsim mouse
Type RNASEQ
Target gene
Project ARCHS4

Ageing is the biggest risk factor to cardiovascular health and is associated with increased incidence of cardiovascular disease. Cellular senescence, a process driven in part by telomere shortening has been implicated in age-related cardiac dysfunction. However, the role of cellular senescence and its underlying mechanisms in slowly dividing/post-mitotic cardiomyocytes is not understood. SOURCE: Neil,Alistair,Robertson (neil.alistair.robertson@hotmail.co.uk) - P. Adams University of Glasgow

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